PeptaHub
The comprehensive peptide reference
COGNITIVEPEPTIDE PROFILE

Noopept

Also known as GVS-111, Omberacetam, N-phenylacetyl-L-prolylglycine ethyl ester

Noopept (INN: Omberacetam) is a synthetic dipeptide derivative developed in Russia by the Zakusov Research Institute of Pharmacology. Its chemical structure is N-phenylacetyl-L-prolyl-glycine ethyl ester, representing the ethyl ester prodrug of the endogenous cycloprolylglycine (CPG) dipeptide. Effective at doses approximately 1000-fold lower than piracetam by weight, it is among the most potent studied nootropic compounds. It was developed for cognitive impairment associated with aging, vascular disorders, and traumatic brain injury.

Last updated April 10, 2026

TL;DR

Quick summary

Noopept (Omberacetam) is a synthetic dipeptide derivative roughly 1000-fold more potent than piracetam. It acts via AMPA receptor modulation and BDNF/NGF upregulation, registered as a prescription drug in Russia though no FDA-regulated trials exist.

§ 01

Overview

Noopept (INN: Omberacetam) is a synthetic dipeptide derivative developed in Russia by the Zakusov Research Institute of Pharmacology. Its chemical structure is N-phenylacetyl-L-prolyl-glycine ethyl ester, representing the ethyl ester prodrug of the endogenous cycloprolylglycine (CPG) dipeptide. Effective at doses approximately 1000-fold lower than piracetam by weight, it is among the most potent studied nootropic compounds. It was developed for cognitive impairment associated with aging, vascular disorders, and traumatic brain injury.

§ 02

Mechanism of action

After oral absorption, Noopept is hydrolyzed to its active metabolite cycloprolylglycine (CPG), which modulates AMPA-type glutamate receptors and facilitates long-term potentiation in hippocampal circuits. Noopept also increases cerebral NGF (nerve growth factor) and BDNF expression, supporting neuronal survival and synaptic density. Additional mechanisms include modulation of voltage-dependent calcium channels and calcium-activated potassium channels, regulating neuronal excitability. Research has identified HIF-1 (hypoxia-inducible factor-1) DNA-binding activity as another proposed primary mechanism, potentially providing neuroprotection under hypoxic or ischemic conditions. Antioxidant activity via inhibition of intracellular reactive oxygen species accumulation contributes to its neuroprotective profile.

§ 03

Dosing protocols

PurposeRouteDosageFrequency
cognitive enhancement researchoral1030 mgtwice daily

Dosing information is for educational purposes only. Consult a qualified healthcare professional before using any peptide.

§ 04

Research summary

Russian preclinical studies across multiple animal models document cognitive enhancement, reduced anxiety, and neuroprotection against ischemia, oxidative stress, and amyloid toxicity. A PMC-indexed study (GVS-111 prevents oxidative damage in Down's syndrome neurons) demonstrated dose-dependent neuroprotection with IC50 of 1.21 μM, significantly outperforming piracetam and vitamin E antioxidants. The compound displays oral bioavailability with confirmed brain penetration. Russian clinical trials in patients with mild cognitive impairment showed improvements in memory and attention; however, these trials have not been replicated under ICH-GCP standards. No FDA-regulated clinical trials have been completed as of 2026.[1][2][3][4]

📄This section cites 4 peer-reviewed sources. View all references →
§ 04b

Evidence grading

Each claimed benefit is graded by the strength of available evidence. Grades reflect study quality, not effect size.

preliminary
Enhances memory/cognitionRussian clinical trials in mild cognitive impairment show memory/attention gains; not ICH-GCP replicated
preliminary
Upregulates NGF and BDNFRat hippocampus study (Bull Exp Biol Med 2009) shows NGF/BDNF mRNA upregulation
preliminary
Neuroprotection against oxidative stressGVS-111 study showed IC50 1.21 μM dose-dependent neuroprotection vs oxidative damage
preliminary
Attenuates tau hyperphosphorylation2014 J Biomed Sci AD cellular model showed apoptosis and tau reduction in vitro

Strong = multiple RCTs · Moderate = limited trials or observational · Preliminary = animal or in vitro only · Insufficient = anecdotal or no published data

§ 05

Side effects

Headache (common, especially without choline supplementation)
Irritability at high doses
Brain fog or fatigue
Sleep disturbance if dosed late
Rare: nausea

Side effects vary by individual. This is not an exhaustive list. Report unusual symptoms to a healthcare professional.

§ 06

Common stacks

Peptides commonly paired with Noopept for synergistic effects.

§ 08

Sourcing & access

Research compound

Noopept is classified as a research compound. Regulatory status varies by jurisdiction. Always verify current legal status and source from vendors providing third-party certificates of analysis (COA).

§ 09

Frequently asked questions

Noopept (Omberacetam, GVS-111) is a synthetic dipeptide derivative developed in Russia as a prodrug of cycloprolylglycine. It is one of the most potent studied nootropics, effective at doses approximately 1000-fold lower than piracetam by weight.

After oral absorption, Noopept is hydrolyzed to cycloprolylglycine (CPG), which modulates AMPA glutamate receptors and facilitates long-term potentiation in hippocampal circuits. It also increases NGF and BDNF expression, modulates calcium and potassium channels, and activates HIF-1 DNA-binding for neuroprotection under hypoxic conditions.

Common side effects include headache (especially without choline supplementation), irritability at high doses, brain fog or fatigue, and sleep disturbance if dosed late. It is a registered prescription drug in Russia but is not FDA-approved; it is unscheduled in the UK and many other jurisdictions.

The standard oral dose is 10 to 30 mg daily, starting at 10 mg once daily. The recommended cycle is 56 days on followed by 4 weeks off. Sublingual or intranasal routes are reported as faster-onset alternatives.

§ 10

Research references

  1. Neuroprotective properties of nootropic dipeptide GVS-111 in in vitro oxygen-glucose deprivation, glutamate toxicity and oxidative stressAndreeva NA, Stel'mashuk EV, Isaev NK, et al.Bull Exp Biol Med, 2000PubMed
  2. Noopept stimulates the expression of NGF and BDNF in rat hippocampusOstrovskaya RU, Gudasheva TA, Zaplina AP, et al.Bull Exp Biol Med, 2009PubMed
  3. Neuroprotective effect of novel cognitive enhancer noopept on AD-related cellular model involves the attenuation of apoptosis and tau hyperphosphorylationJia X, Gharibyan AL, Öhman A, et al.J Biomed Sci, 2014PubMed
  4. Proline-containing dipeptide GVS-111 retains nootropic activity after oral administrationGudasheva TA, Boyko SS, Ostrovskaya RU, et al.Eur J Drug Metab Pharmacokinet, 2002PubMed
● READER REVIEWS

What readers say about Noopept

No reader reviews yet. If you’ve used Noopept, share your experience — your review helps the next person decide.