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WEIGHT LOSSPEPTIDE PROFILE

Survodutide

Also known as BI 456906, BI-456906

Survodutide (BI 456906) is a dual glucagon receptor (GCGR) and GLP-1 receptor (GLP-1R) agonist developed by Boehringer Ingelheim and Zealand Pharma. It is in Phase 3 trials for obesity and overweight (SYNCHRONIZE program) and received FDA Fast Track Designation for metabolic dysfunction-associated steatohepatitis (MASH/NASH). The dual mechanism adds hepatic fat clearance and energy expenditure on top of appetite suppression.

Last updated April 10, 2026

TL;DR

Quick summary

Survodutide is Boehringer Ingelheim and Zealand Pharma's dual glucagon/GLP-1 receptor agonist in Phase 3 trials for obesity and MASH. It has received FDA Fast Track designation for MASH and is not yet approved.

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Overview

Survodutide (BI 456906) is a dual glucagon receptor (GCGR) and GLP-1 receptor (GLP-1R) agonist developed by Boehringer Ingelheim and Zealand Pharma. It is in Phase 3 trials for obesity and overweight (SYNCHRONIZE program) and received FDA Fast Track Designation for metabolic dysfunction-associated steatohepatitis (MASH/NASH). The dual mechanism adds hepatic fat clearance and energy expenditure on top of appetite suppression.

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Mechanism of action

Survodutide simultaneously activates two distinct receptor pathways. GLP-1 receptor agonism slows gastric emptying, enhances glucose-dependent insulin secretion, and reduces appetite via central hypothalamic signaling — effects shared with semaglutide and tirzepatide. The added glucagon receptor (GCGR) agonism introduces direct hepatic mechanisms not present in GLP-1-only agents: stimulation of mitochondrial fatty acid beta-oxidation, upregulation of energy expenditure through farnesoid X receptor (FXR) signaling, and induction of fibroblast growth factor 21 (FGF21) secretion. FGF21 is a key metabolic hormone that increases thermogenesis and improves insulin sensitivity. Together, the dual agonism increases both energy expenditure and reduces caloric intake, producing weight loss that may exceed GLP-1-only agents in certain populations.

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Dosing protocols

PurposeRouteDosageFrequency
Obesity / weight management (Phase 3 trial)subcutaneous0.64.8 mgOnce weekly

Dosing information is for educational purposes only. Consult a qualified healthcare professional before using any peptide.

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Research summary

Phase 2 data showed mean weight loss ranging from 6.2% (0.6 mg) to 14.9% (4.8 mg) at 46 weeks vs 2.8% for placebo. In the Phase 2 MASH trial, up to 62% of patients at 4.8 mg achieved histological MASH improvement without worsening fibrosis, vs 14% placebo. Phase 3 SYNCHRONIZE-1 (obesity without T2D) and SYNCHRONIZE-2 (obesity with T2D) trials are ongoing globally, with SYNCHRONIZE-CVOT evaluating long-term cardiovascular safety. Results pending as of 2026.[1][2][3]

📄This section cites 3 peer-reviewed sources. View all references →
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Evidence grading

Each claimed benefit is graded by the strength of available evidence. Grades reflect study quality, not effect size.

moderate
Weight loss via GLP-1/glucagon dual agonismJastreboff Lancet Diabetes Endocrinol 2024 (Phase 2): 6.2-14.9% weight loss at 46 weeks across doses; dose-response confirmed
moderate
MASH/NASH liver improvementSanyal NEJM 2024 Phase 2 MASH trial: 62% histological improvement at 4.8 mg vs 14% placebo; FDA Fast Track designation
preliminary
Phase 3 obesity efficacySYNCHRONIZE-1 baseline characteristics published Obesity 2025 (Kushner); pivotal outcomes pending as of 2026
preliminary
FGF21-mediated thermogenesis increaseEstablished glucagon receptor biology; mechanism supported by preclinical and Phase 1 PD markers; not isolated in human outcomes
insufficient
Cardiovascular safety and outcomesSYNCHRONIZE-CVOT ongoing; no completed cardiovascular outcomes trial as of 2026

Strong = multiple RCTs · Moderate = limited trials or observational · Preliminary = animal or in vitro only · Insufficient = anecdotal or no published data

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Side effects

Nausea
Vomiting
Diarrhea
Constipation
Decreased appetite
Injection site reactions
Fatigue

Side effects vary by individual. This is not an exhaustive list. Report unusual symptoms to a healthcare professional.

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Common stacks

Peptides commonly paired with Survodutide for synergistic effects.

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Sourcing & access

Research compound

Survodutide is classified as a research compound. Regulatory status varies by jurisdiction. Always verify current legal status and source from vendors providing third-party certificates of analysis (COA).

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Frequently asked questions

Survodutide (BI 456906) is a dual glucagon receptor and GLP-1 receptor agonist developed by Boehringer Ingelheim and Zealand Pharma. It is in Phase 3 trials for obesity and has received FDA Fast Track designation for metabolic dysfunction-associated steatohepatitis.

Survodutide simultaneously activates two receptors. GLP-1 receptor agonism slows gastric emptying and suppresses appetite, while glucagon receptor agonism stimulates hepatic fatty acid oxidation, increases energy expenditure, and induces FGF21 secretion to enhance thermogenesis and insulin sensitivity.

No. Survodutide is an investigational compound in Phase 3 clinical trials and is not approved for human use outside of those trials. It is available only through authorized research programs.

Phase 2 data showed mean weight loss ranging from 6.2% at 0.6 mg to 14.9% at 4.8 mg at 46 weeks, versus 2.8% for placebo. In a Phase 2 MASH trial, up to 62% of patients at 4.8 mg achieved histological MASH improvement without worsening fibrosis versus 14% on placebo.

Phase 3 SYNCHRONIZE protocols titrate from 0.6 mg once weekly subcutaneously up to a maximum studied dose of 4.8 mg, consistent with the dose-finding range from Phase 2 trials. The approximately one-week half-life supports once-weekly administration. Survodutide is not available outside authorized clinical trials and cannot be obtained through compounding or research chemical channels.

Reported side effects in Phase 2 and Phase 3 trials include nausea, vomiting, diarrhea, constipation, decreased appetite, injection site reactions, and fatigue. The profile is consistent with the broader GLP-1 class, and the glucagon component has not produced significant hepatic or glycemic safety signals in the clinical dose range studied so far. Long-term cardiovascular safety remains under evaluation in SYNCHRONIZE-CVOT.

Both are dual-mechanism weight loss peptides in clinical development, but they activate different receptor pairs. Tirzepatide is a GLP-1/GIP dual agonist, while survodutide is a GLP-1/glucagon dual agonist that additionally stimulates hepatic fat clearance and energy expenditure.

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Research references

  1. Glucagon and GLP-1 receptor dual agonist survodutide for obesity: a randomised, double-blind, placebo-controlled, dose-finding phase 2 trialJastreboff AM, Kaplan LM, Frías JP, et al.Lancet Diabetes & Endocrinology, 2024PubMed
  2. A Phase 2 Randomized Trial of Survodutide in MASH and FibrosisSanyal AJ, Bedossa P, Fraessdorf M, et al.New England Journal of Medicine, 2024PubMed
  3. Survodutide for treatment of obesity: Baseline characteristics of participants in a randomized, double-blind, placebo-controlled, phase 3 trial (SYNCHRONIZE-1)Kushner RF, Schwartz AL, Perreault L, et al.Obesity, 2025PubMed
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