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OTHERPEPTIDE PROFILE

Substance P

Also known as SP, Neurokinin P, substance P undecapeptide

Substance P is an 11-amino acid neuropeptide belonging to the tachykinin family, with the sequence Arg-Pro-Lys-Pro-Gln-Gln-Phe-Phe-Gly-Leu-Met-NH2. It functions as both a neurotransmitter and neuromodulator in the central and peripheral nervous systems, playing a key role in pain signaling, neurogenic inflammation, and mood regulation. It was among the first neuropeptides discovered and has been studied for over 90 years.

Last updated April 10, 2026

TL;DR

Quick summary

Substance P is an 11-amino acid tachykinin neuropeptide central to pain signaling, neurogenic inflammation, and mood regulation. NK1 receptor antagonists derived from Substance P research (aprepitant, netupitant) are FDA-approved antiemetics, validating the SP-NK1R axis as a therapeutic target.

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Overview

Substance P is an 11-amino acid neuropeptide belonging to the tachykinin family, with the sequence Arg-Pro-Lys-Pro-Gln-Gln-Phe-Phe-Gly-Leu-Met-NH2. It functions as both a neurotransmitter and neuromodulator in the central and peripheral nervous systems, playing a key role in pain signaling, neurogenic inflammation, and mood regulation. It was among the first neuropeptides discovered and has been studied for over 90 years.

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Mechanism of action

Substance P acts primarily through the NK1 (neurokinin-1) receptor, a Gq-coupled GPCR, with lower affinity for NK2 and NK3. Binding to NK1R activates phospholipase C, raises intracellular calcium, and activates PKC, promoting neuronal excitation and synaptic facilitation. In primary afferent nociceptors, SP co-localizes with glutamate and CGRP, facilitating pain transmission to the dorsal horn. Peripheral SP release from afferent nerve endings triggers mast cell degranulation, vasodilation, plasma extravasation, and immune cell recruitment — the hallmarks of neurogenic inflammation.

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Dosing protocols

PurposeRouteDosageFrequency
nociception research (animal)intravenous110 nmol/kgper study protocol

Dosing information is for educational purposes only. Consult a qualified healthcare professional before using any peptide.

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Research summary

NK1 receptor antagonists (aprepitant, netupitant) have been developed and are FDA-approved as antiemetics in chemotherapy-induced nausea/vomiting, validating the SP-NK1R axis as a therapeutic target. Research into NK1 antagonists for depression, PTSD, and chronic pain continues. Elevated SP levels are associated with fibromyalgia, IBS, and inflammatory arthritis. SP also plays a role in wound healing and angiogenesis, creating therapeutic interest beyond pain.[1][2][3][4][5]

📄This section cites 5 peer-reviewed sources. View all references →
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Evidence grading

Each claimed benefit is graded by the strength of available evidence. Grades reflect study quality, not effect size.

strong
NK1 antagonists FDA-approved for CINVAprepitant and netupitant are FDA-approved antiemetics validating the SP-NK1R axis in chemotherapy nausea
moderate
Facilitates spinal pain transmissionDecades of preclinical evidence show SP co-released with glutamate and CGRP in nociceptors drives dorsal-horn signaling
moderate
Drives neurogenic inflammation peripherallyEstablished animal and in vitro data show NK1R-mediated mast cell degranulation and plasma extravasation
preliminary
Elevated in fibromyalgia and IBSObservational case-control studies report elevated CSF/plasma SP in fibromyalgia and inflammatory conditions
preliminary
Promotes wound healing via NK1RAnimal and in vitro evidence suggests SP supports angiogenesis and tissue repair; no human trials

Strong = multiple RCTs · Moderate = limited trials or observational · Preliminary = animal or in vitro only · Insufficient = anecdotal or no published data

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Side effects

Vasodilation and flushing (IV research doses)
Bronchoconstriction (high doses)
Pain at injection site

Side effects vary by individual. This is not an exhaustive list. Report unusual symptoms to a healthcare professional.

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Common stacks

Peptides commonly paired with Substance P for synergistic effects.

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Sourcing & access

Research compound

Substance P is classified as a research compound. Regulatory status varies by jurisdiction. Always verify current legal status and source from vendors providing third-party certificates of analysis (COA).

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Frequently asked questions

Substance P is an 11-amino acid neuropeptide belonging to the tachykinin family that functions as both a neurotransmitter and neuromodulator. It plays key roles in pain signaling, neurogenic inflammation, and mood regulation in the central and peripheral nervous systems.

Substance P acts primarily through the NK1 (neurokinin-1) receptor, a Gq-coupled GPCR. Binding activates phospholipase C, raises intracellular calcium, and promotes neuronal excitation. Peripheral SP release triggers mast cell degranulation, vasodilation, and immune cell recruitment, the hallmarks of neurogenic inflammation.

Substance P is a research compound not approved for human administration. In research settings, intravenous doses can cause vasodilation, flushing, bronchoconstriction at high doses, and pain at injection sites. NK1 receptor antagonists targeting this pathway have well-established clinical safety profiles.

NK1 receptor antagonists such as aprepitant and netupitant are FDA-approved for chemotherapy-induced nausea and vomiting. Research into NK1 antagonists for depression, PTSD, and chronic pain continues, with elevated Substance P levels observed in fibromyalgia, IBS, and inflammatory arthritis.

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Research references

  1. Biochemistry, Substance PKarpel-Massler G, Siegelin MD, et al.StatPearls, 2020Review
  2. Substance P, a Neuropeptide, Promotes Wound Healing via Neurokinin-1 ReceptorMashaghi A, Marmalidou A, et al.Frontiers in Medicine, 2021PubMed
  3. Inflammation and Organ Injury the Role of Substance P and Its ReceptorsYong YK, Tan JJ, et al.Current Drug Targets, 2023Review
  4. The Role of Substance P Within Traumatic Brain Injury and Implications for TherapyDonkin JJ, Vink R, et al.Frontiers in Neurology, 2023PubMed
  5. Modulating the tachykinin: Role of substance P and neurokinin receptor expression in ocular surface disordersNishida T, Yanai R, et al.The Ocular Surface, 2022PubMed
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