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OTHERPEPTIDE PROFILE

Buserelin

Also known as Suprefact, Suprecur, Profact, Receptal

Buserelin is a synthetic GnRH agonist peptide widely approved in Europe, Canada, and other regions under brand names including Suprefact and Suprecur for prostate cancer, endometriosis, and female infertility. It is not approved in the United States but is a first-line hormonal therapy in many countries. Available as subcutaneous injection, nasal spray, or implant, it achieves sex steroid suppression equivalent to surgical castration through pituitary GnRH receptor desensitization.

Last updated April 10, 2026

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Buserelin: quick citable summary

Buserelin is listed by PeptaHub as a other peptide with a prescription legal-status classification. The page summarizes mechanism, research context, common routes, safety notes, and references for writers and AI answer engines.

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PeptaHub. “Buserelin: Mechanism, Research Context, Safety.” peptahub.com, 2026. https://peptahub.com/peptides/buserelin. Licensed CC BY 4.0.

License: Creative Commons Attribution 4.0 International. Link back to https://peptahub.com/peptides/buserelin.

SAMEAS / EXTERNAL IDS
Buserelin CAS: 57982-77-1
QUICK ANSWER

What is Buserelin?

Buserelin (Suprefact) is a GnRH superagonist 20-170x more potent than native GnRH, approved in Europe and Canada for prostate cancer, endometriosis, and IVF. It suppresses sex steroids to castrate levels via pituitary desensitization.

§ 01

Overview

Buserelin is a synthetic GnRH agonist peptide widely approved in Europe, Canada, and other regions under brand names including Suprefact and Suprecur for prostate cancer, endometriosis, and female infertility. It is not approved in the United States but is a first-line hormonal therapy in many countries. Available as subcutaneous injection, nasal spray, or implant, it achieves sex steroid suppression equivalent to surgical castration through pituitary GnRH receptor desensitization.

§ 02

Mechanism of action

Buserelin is a GnRH superagonist with approximately 20–170 times the potency of endogenous GnRH for stimulating LH and FSH secretion. Structural modifications — D-serine(tBu) at position 6 and ethylamide at the C-terminus — confer enzymatic stability and high receptor binding affinity. Initial administration triggers an LH/FSH flare response. With continuous exposure (unlike pulsatile endogenous GnRH), buserelin maintains constant GnRH receptor occupancy, inducing receptor downregulation and uncoupling. Within 2–4 weeks, pituitary gonadotrophs become desensitized, LH and FSH secretion collapses, and gonadal testosterone or estradiol falls to castrate levels. In prostate cancer, androgen withdrawal inhibits androgen receptor signaling that drives tumor proliferation.

§ 03

Reported study ranges

PurposeRouteReported rangeFrequency
Prostate cancer — ADTsubcutaneous500500 mcgthree times daily for 7 days, then nasal maintenance
Endometriosis / IVF down-regulationnasal300900 mcgtwo to three times daily

Reported ranges are for research context only. Consult a qualified healthcare professional before using any peptide.

Convert Buserelin research-range units

Need to convert mg to mcg, dose volume, or U-100 syringe units? Use the peptide dose unit converter for educational calculation support.

§ 04

Research summary

European clinical trials established buserelin as effective ADT for advanced prostate cancer, achieving castrate testosterone in >95% of patients within 4 weeks. Long-term (3-year) data demonstrate disease stabilization comparable to orchiectomy. For endometriosis, 6-month nasal spray or implant treatment reduces lesion extent and pain scores significantly. In IVF protocols, buserelin down-regulation is used in the long protocol to synchronize follicular development, though GnRH antagonists are increasingly preferred due to shorter treatment duration. Buserelin's clinical profile is similar to leuprolide and other GnRH agonists in its class.[1][2][3][4]

📄This section cites 4 peer-reviewed sources. View all references →
§ 04b

Evidence grading

Each claimed benefit is graded by the strength of available evidence. Grades reflect study quality, not effect size.

strong
Castrate testosterone in prostate cancerKlijn 1985 Lancet RCT vs orchiectomy; >95% of patients reach castrate testosterone within 4 weeks
strong
Endometriosis lesion and pain reductionCochrane 2007 meta-analysis of GnRH agonists confirms efficacy in endometriosis over 6-month courses
strong
IVF pituitary downregulationHughes 1992 Fertility & Sterility and multiple RCTs establish long-protocol IVF downregulation efficacy
moderate
3-year disease stabilization equals orchiectomyLong-term comparative data in prostate cancer show equivalence to surgical castration

Strong = multiple RCTs · Moderate = limited trials or observational · Preliminary = animal or in vitro only · Insufficient = anecdotal or no published data

§ 05

Side effects

Hot flashes
Sexual dysfunction / decreased libido
Initial disease flare (prostate cancer)
Bone mineral density loss (long-term)
Nasal irritation (nasal spray formulation)
Headache
Fatigue
Mood changes
Injection site reactions (subcutaneous formulation)

Side effects vary by individual. This is not an exhaustive list. Report unusual symptoms to a healthcare professional.

§ 06

Common stacks

Peptides commonly paired with Buserelin for synergistic effects.

§ 08

Sourcing & access

Prescription required

Buserelin is an FDA-approved prescription medication available through licensed healthcare providers, pharmacies, and label-appropriate access programs; compounded access depends on current FDA shortage status and compounding rules.

§ 09

Frequently asked questions

Buserelin is a synthetic GnRH agonist peptide approved in Europe, Canada, and other regions under brand names Suprefact and Suprecur for prostate cancer, endometriosis, and female infertility. It is not approved in the United States.

Buserelin is a GnRH superagonist 20 to 170 times more potent than native GnRH. Initial administration triggers an LH/FSH flare, but continuous exposure causes receptor downregulation and pituitary desensitization, suppressing sex steroids to castrate levels within 2 to 4 weeks.

Buserelin is available as subcutaneous injection for induction phases, nasal spray for maintenance and down-regulation, and implant formulations for sustained release. The subcutaneous route is typically used for the first 7 days of prostate cancer therapy, while the nasal spray at 200 to 400 mcg per nostril is commonly used for endometriosis and IVF down-regulation protocols.

Common side effects include hot flashes, sexual dysfunction, initial disease flare in prostate cancer, bone mineral density loss with long-term use, nasal irritation from the spray, headache, fatigue, and mood changes.

§ 10

Research references

  1. Buserelin GnRH agonist in prostate cancer: clinical pharmacology and efficacyBorgmann V, Hardt W, et al.Lancet, 1982PubMed
  2. Buserelin versus orchiectomy in advanced prostate cancer: randomized trialKlijn JG, de Jong FH, et al.Lancet, 1985PubMed
  3. GnRH agonists in endometriosis: meta-analysis of efficacySelak V, Farquhar C, et al.Cochrane Database Syst Rev, 2007PubMed
  4. Buserelin IVF protocol: pituitary downregulation and ovarian stimulationHughes EG, Fedorkow DM, et al.Fertil Steril, 1992PubMed
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